Traumatic brain injury Treatments and drugs. Mild injury Mild traumatic brain injuries usually require no treatment other than rest and over-the-counter pain relievers to treat a headache.
However, a person with a mild traumatic brain injury usually needs to be monitored closely at home for any persistent, worsening or new symptoms. He or she also may have follow-up doctor appointments. The doctor will indicate when a return to work, school or recreational activities is appropriate. It's best to avoid physical or thinking (cognitive) activities that make things worse until symptoms have resolved. Immediate emergency care Emergency care for moderate to severe traumatic brain injuries focuses on making sure the person has an adequate oxygen and blood supply, maintaining blood pressure, and preventing any further injury to the head or neck.
People with severe injuries may also have other injuries that need to be addressed. Medications Medications to limit secondary damage to the brain immediately after an injury may include: TBI: Treatments for Traumatic Brain Injury. White-matter structure in the right hemisphere predicts Mandarin Chinese learning success. The effects of bilingualism on the white matter structure of the brain.
Author Affiliations Edited by Patricia K.
Kuhl, University of Washington, Seattle, WA, and approved December 12, 2014 (received for review July 29, 2014) Significance This article fills an important gap in the literature on structural changes in the brain that are induced by speaking two languages. An fMRI study of language lateralization in children and adults. The ontogenesis of language lateralization and its relation to handedness. Structural brain changes related to bilingualism: does immersion make a difference?
Introduction Experience-dependent changes in brain structure were first investigated in rodents placed in environmentally enriched versus very sparsely equipped standard cages.
These early animal studies reported effects of environmental enrichment on brain weight (Rosenzweig et al., 1962) or cortical thickness (Rosenzweig et al., 1972) suggesting a structural adaptation process of the brain in response to experience. Since then, experience-dependent changes in human brain structure have been investigated in relation to various learning experiences, ranging from complex visuo-motor tasks like juggling to musical proficiency as well as to various aspects of language learning (see, e.g., May, 2011; Zatorre et al., 2012; Lovden et al., 2013). The perineuronal net and the control of CNS plasticity. Disruption of the perineuronal net in the hippocampus or medial prefrontal cortex impairs fear conditioning. Abstract The perineuronal net (PNN) surrounds neurons in the central nervous system and is thought to regulate developmental plasticity.
A few studies have shown an involvement of the PNN in hippocampal plasticity and memory storage in adult animals. In addition to the hippocampus, plasticity in the medial prefrontal cortex (mPFC) has been demonstrated to be critical for the storage of long-term memory, particularly memories for temporally separated events. U.pdf. Cerebral asymmetry and language development: cause, correlate or consequence? IDA_DSM-5%20Changes.pdf. Post-traumatic stress disorder and declarative memory functioning: a review. Post-Traumatic Stress Disorder (PTSD) What is post-traumatic stress disorder, or PTSD?
PTSD is an anxiety disorder that some people get after seeing or living through a dangerous event. When in danger, it’s natural to feel afraid. This fear triggers many split-second changes in the body to prepare to defend against the danger or to avoid it. Deficits in short-term memory in adult survivors of childhood abuse. Untitled. Attention, Learning, and Memory in Posttraumatic Stress Disorder. Specific%20Learning%20Disorder%20Fact%20Sheet.pdf. Does PTSD Cause Learning Disabilities? Specific Learning Disorder. This developmental disorder involves difficulties learning and using academic skills.
“Specific Learning Disorder” has become the umbrella term for mathematics, reading, and written expression disorders in the updated DSM-5. The DSM-IV previously classified these as separate diagnoses. Instead, these disorders are now housed under one diagnosis with added specifiers (e.g., specific learning disorder with impaired reading). The biological origin of a learning disorder is likely an interaction of genetic and environmental factors, which affect the brain’s ability to perceive or process verbal or nonverbal information efficiently and accurately.
Key academic skills of deficit include reading of single words accurately and fluently, reading comprehension, written expression and spelling, arithmetic calculation, and mathematical reasoning (solving math problems). A key feature is that the individual’s performance in a particular area is well below average for age. 1. Role of MRI in Epilepsy. 00_Nutt_McLellan.pdf. Uncovering the Hidden Risk Architecture of the Schizophrenias: Confirmation in Three Independent Genome-Wide Association Studies. Complex diseases, such as schizophrenia, may be influenced by hundreds or thousands of genetic variants that interact with one another in complex ways, and consequently display a multifaceted genetic architecture (1).
The genetic architecture of heritable diseases refers to the number, frequency, and effect sizes of genetic risk alleles and the way they are organized into genotypic networks (2). In complex disorders, the same genotypic networks may lead to different clinical outcomes (a concept known as multifinality, which is called pleiotropy in genetics), and different genotypic networks may lead to the same clinical outcome (equifinality, which is also described as heterogeneity) (1, 3). Autism Social and emotional adjustment o.'.. [J Child Psychol Psychiatry. 2004.
PTSD. HIV. Epilepsy. Traumatic Brain Injury: Rehabilitation Neurology. Developmental Plasticity and Evolution - Mary Jane West-Eberhard. Encephalomalacia. Conduct Disorders. How ADHD Puts Athletes In The Zone. ADHD. Autism. Schizophrenia. Math Disorders Neuropsychology. NeuroPsy PTSD. Language Disorders. Autism. Stroke & Infarct. Turner's Syndrome. Cell Phone Radiation. Cancer & Treatment Effects. Researchers discover new drugs to combat the root cause of multiple sclerosis. New research published this week in Nature has found several drugs could lead to new treatment options for multiple sclerosis (MS), including two drugs that effectively treat MS at the source, in vivo.
When administered at the peak of disease, these two drugs showed a striking reversal of disease severity. At the pathological level, MS is a disease in which the immune system attacks the protective myelin sheath, a type of insulation that covers nerves, ultimately disrupting communication between the brain and the body and leading to nerve deterioration.
To prevent neural degeneration requires remyelination through new oligodendrocytes, which create the myelin sheath. These two drugs, miconazole and clobetasol, were found to treat the source of the problem by reversing this process, increasing the number of new oligodendrocytes and enhancing remyelination. "Current therapies focus on stopping immune system attacks, slowing the progression of the disease. Psychological Medicine - Abstract - The neuropsychology of antisocial personality disorder. The-neurobiology-of-antisocial-personality-disorder.pdf. Brain hyper-connectivity and operation-specific deficits during ari... Stress acts cumulatively to precipitate Alzheimer... [J Neurosci. 2011. Slide Show: The Neural Control of Visually Guided Eye Movements A-1. Sickle Cell and the Brain.
Diagnostic Testing, Mimics of Stroke and Stroke Prevention Robert J. Adams, MS, MD * Neurofibromatosis — Why choose Mayo Clinic. Neurofibromatosis is a genetic disorder that disturbs cell growth in your nervous system, causing tumors to form on nerve tissue.
These tumors may develop anywhere in your nervous system, including in your brain, spinal cord and nerves. Neurofibromatosis is usually diagnosed in childhood or early adulthood. The tumors are usually noncancerous (benign), but in some cases these tumors become cancerous (malignant) tumors. People with neurofibromatosis often experience only mild symptoms. International Craniofacial Institute - Cleft Lip and Treatment Center. Tumors: Neurofibromatosis 01 Also known as von Recklinghausen disease, neurofibromatosis is rarely cancerous. Neurofibromatosis grows around the nerve cells/sheaths and affects all nearby tissues: skin, the layer below the skin (subcutaneous) and bone. Typically, neurofibromatosis is first noticed as patches of "café au lait" colored areas of skin which look like birthmarks. Then, the neurofibromatosis nodules themselves are noted. Human biology - How does the brain's energy consumption depend on mental activity? - Biology Beta - Stack Exchange.
I answered on the facts of this question already on skeptics.SE, here and here. You should read both papers very carefully, I highlighted the most important facts but this is a very tricky question, esp. when it comes to defining what mental activitiy is. The papers also give an explanation of how fMRI signal is linked to NEURONAL activity, as far as I remember there is no strong direct link.
You assume in your question that a mathematician solving a differential equation needs higher mental activity than a child reading a book. Is this legitimate? It seems intuitive but also very subjective.