Learning Mind - Expand your mind with lifelong learning. RGS14. Regulator of G-protein signaling 14 (RGS14) is a protein that in humans is encoded by the RGS14 gene. Function RGS14 is a member of the regulator of G protein signalling family.
This protein contains one RGS domain, two Raf-like Ras-binding domains (RBDs), and one GoLoco motif. The protein attenuates the signaling activity of G-proteins by binding, through its GoLoco domain, to specific types of activated, GTP-bound G alpha subunits. Acting as a GTPase activating protein (GAP), the protein increases the rate of conversion of the GTP to GDP. Increasing the expression of the RGS14 protein in the V2 secondary visual cortex of mice promotes the conversion of short-term to long-term object-recognition memory. Conversely RGS14 is enriched in CA2 pyramidal neurons and suppresses synaptic plasticity of these synapses and hippocampal-based learning and memory. Interactions RGS14 has been shown to interact with: References Further reading
Wellcome: Axon Game. What Your Facebook Account Says About Your Brain. Why do some people have thousands of friends on Facebook and others have just a few?
It turns out that it depends on the size of their brain. A recent study found that people with more friends have a larger orbital prefrontal cortex, the region of the brain in your forehead right above your eyes. This brain region is involved in complex cognitive processes such as thinking about oneself and thinking about what other people might be thinking. Other recent studies found that people with larger social networks (including the number of friends on Facebook) also have a larger amygdala (a brain region involved in emotion regulation). Establishing and maintaining many social relationships requires a great deal of brainpower. Take a primate species like the rhesus macaque for example. There is an interesting twist to the Machiavellian Intelligence theory of brain evolution. Powell J., Lewis P.A., Roberts, N., García-Fiñana, M, & Dunbar, R.I.M. 2012. Bickart, K. Multitasking:This is your Brain on Media.
Embed the above image on your site <a href=" src=" border="0" alt="Rasmussen College" /></a> External links provided on Rasmussen.edu are for reference only.
Rasmussen College does not guarantee, approve, control, or specifically endorse the information or products available on websites linked to, and is not endorsed by website owners, authors and/or organizations referenced. The Limits of Intelligence. Santiago Ramón y Cajal, the Spanish Nobel-winning biologist who mapped the neural anatomy of insects in the decades before World War I, likened the minute circuitry of their vision-processing neurons to an exquisite pocket watch.
He likened that of mammals, by comparison, to a hollow-chested grandfather clock. Indeed, it is humbling to think that a honeybee, with its milligram-size brain, can perform tasks such as navigating mazes and landscapes on a par with mammals. A honeybee may be limited by having comparatively few neurons, but it surely seems to squeeze everything it can out of them. At the other extreme, an elephant, with its five-million-fold larger brain, suffers the inefficiencies of a sprawling Mesopotamian empire. Signals take more than 100 times longer to travel between opposite sides of its brain—and also from its brain to its foot, forcing the beast to rely less on reflexes, to move more slowly, and to squander precious brain resources on planning each step. Brain-based learning, ideas, and materials. The Whole Brain Atlas.
Focus on Brain Disorders. The brain is the most complex organ in the human body, in terms of both its structure and chemical composition. Created by our genes, life experiences and the environment around us, the brain acquires, coordinates and disseminates information to control how we think, behave, learn and feel. To do this, all of the one hundred billion cells in this complex organ must effectively communicate with each other, and failure to do so may cause or contribute to brain dysfunction and mental illness.
Factsheet: Overview of mental disorders. How to increase serotonin in the human brain without drugs. For the last 4 decades, the question of how to manipulate the serotonergic system with drugs has been an important area of research in biological psychiatry, and this research has led to advances in the treatment of depression.
Research on the association between various polymorphisms and depression supports the idea that serotonin plays a role, not only in the treatment of depression but also in susceptibility to depression and suicide. The research focus here has been on polymorphisms of the serotonin transporter, but other serotonin-related genes may also be involved.1–5 In the future, genetic research will make it possible to predict with increasing accuracy who is susceptible to depression. Much less attention has been given to how this information will be used for the benefit of individuals with a serotonin-related susceptibility to depression, and little evidence exists concerning strategies to prevent depression in those with such a susceptibility.