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Animation of the fusiform area, the area damaged in prosopagnosia. Prosopagnosia /ˌprɒsəpæɡˈnoʊʒə/ (Greek: "prosopon" = "face", "agnosia" = "not knowing"), also called face blindness,[1] is a cognitive disorder of face perception where the ability to recognize faces is impaired, while other aspects of visual processing (e.g., object discrimination) and intellectual functioning (e.g., decision making) remain intact. The term originally referred to a condition following acute brain damage (acquired prosopagnosia), but a congenital or developmental form of the disorder also exists, which may affect up to 2.5% of the population.[2] The specific brain area usually associated with prosopagnosia is the fusiform gyrus,[3] which activates specifically in response to faces. The functionality of the fusiform gyrus allows most people to recognize faces in more detail than they do similarly complex inanimate objects. There are two types of prosopagnosia: acquired and congenital (developmental).

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Scotopic sensitivity syndrome Scotopic sensitivity syndrome (SSS), also known as Visual Stress, Irlen Syndrome, and Asfedia, is a condition relating to the interaction of the central nervous system and the eyes at a physiological level with light. The effects of SSS are most noticeable during activities associated with reading, but an individual with the condition may notice the condition's effects in other activities. The exact cause of SSS is currently under debate within the scientific community. Prosopagnosia Research - About Prosopagnosia What is prosopagnosia? Prosopagnosia (also known as 'face blindness') refers to a severe deficit in recognizing familiar people from their face. While some people report a very selective impairment that only influences the recognition of faces, others find the deficit extends to the recognition of other stimuli, such as objects, cars, or animals.

Recovery from blindness Recovery from blindness is the phenomenon of a blind person gaining the ability to see, usually as a result of medical treatment. As a thought experiment, the phenomenon is usually referred to as Molyneux's Problem. The first published human case was reported in 1728 by the Surgeon William Cheselden. Patients who experience dramatic recovery from blindness experience significant to total agnosia, having serious confusion with their visual perception. List of memory biases In psychology and cognitive science, a memory bias is a cognitive bias that either enhances or impairs the recall of a memory (either the chances that the memory will be recalled at all, or the amount of time it takes for it to be recalled, or both), or that alters the content of a reported memory. There are many different types of memory biases, including: See also[edit]

Visual agnosia Visual agnosia is an impairment in recognition of visually presented objects. It is not due to a deficit in vision (acuity, visual field, and scanning), language, memory, or low intellect.[1] There are two types of visual agnosia: apperceptive agnosia and associative agnosia. Recognition of visual objects occurs at two primary levels. Do you suffer from face blindness? Seven signs and symptoms of prosopagnosia If you read my previous post on the role of cognitive assessment in identifying uniqueness, you’ll know that I’ve worked with a lot of folks who suffer from severe difficulties recognizing faces: a condition known as prosopagnosia or face blindness. I get a lot of emails from people who take the face recognition tests on and want to know what sorts of experiences might indicate that someone has face blindness. If you suspect you have face blindness, you may find you identify with some or many of the experiences below.

Color blindness Color blindness, or color vision deficiency, is the inability or decreased ability to see color, or perceive color differences, under normal lighting conditions. Color blindness affects a significant percentage of the population.[1] There is no actual blindness but there is a deficiency of color vision. The most usual cause is a fault in the development of one or more sets of retinal cones that perceive color in light and transmit that information to the optic nerve. This type of color blindness is usually a sex-linked condition. The genes that produce photopigments are carried on the X chromosome; if some of these genes are missing or damaged, color blindness will be expressed in males with a higher probability than in females because males only have one X chromosome (in females, a functional gene on only one of the two X chromosomes is sufficient to yield the needed photopigments).[2] Background[edit]

Successful Schizophrenia Home | Articles | Resources | Stories | Weller-than-Well | Links | Search Disclaimer: Material found on the Successful Schizophrenia website is for your information only. We are not able dispense specific advice for your situation. If you are under a doctor's care, you should talk with him or her about your mental health goals and if they are not on the same page as you, ask for a referral to a doctor or counselor who is. It may mean interviewing several. If you are on your own, you may wish to contact your local county mental health department to ask for local resources.

Astigmatism (eye) For the more general class of optical aberrations, see Astigmatism. The refractive error of the astigmatic eye stems from a difference in degree of curvature refraction of the two different meridians (i.e., the eye has different focal points in different planes). For example, the image may be clearly focused on the retina in the horizontal plane, but not in the vertical plane. Astigmatism causes difficulties in seeing fine detail resulting in blurred vision. Three options exist for the treatment of astigmatism: spectacles, contact lenses (either hard contact lenses or toric contact lenses), and refractive surgery. Blur from astigmatic lens at different distances

How To Think With Your Hands Monday, June 24, 2013 Over the past few years, while working on my forthcoming book Brilliant, I’ve been watching and taking note as a new conceptualization of intelligence takes shape in the social and biological sciences. This conceptualization involves many lines of inquiry that can be loosely grouped under the title situated cognition: the idea that thinking doesn’t happen in some abstract, disembodied space, but always in a particular brain, in a particular body, located in a particular social and physical world. The moment-by-moment conditions that prevail in that brain, that body, and that world powerfully affect how well we think and perform.

Asthenopia Asthenopia (aesthenopia) from the Greek word "asthen-opia : ασθεν-ωπία" or eye strain is an ophthalmological condition that manifests itself through nonspecific symptoms such as fatigue, pain in or around the eyes, blurred vision, headache and occasional double vision. Symptoms often occur after reading, computer work, or other close activities that involve tedious visual tasks. When concentrating on a visually intense task, such as continuously focusing on a book or computer monitor, the ciliary muscle tightens. This can cause the eyes to get irritated and uncomfortable.

Character Strengths and Virtues In the same way that the Diagnostic and Statistical Manual of Mental Disorders is used to assess and facilitate research on mental disorders, CSV is intended to provide a theoretical framework to assist in developing practical applications for positive psychology.[1] Definition of strengths and virtues[edit] CSV identifies six classes of virtue (i.e., "core virtues"), made up of twenty-four measurable "character strengths".

Associative visual agnosia Inferior view of the brain, depicting the cerebral lobes. Lesions on the occipito-temporal lobes are correlated with associative agnosia. Overview[edit] An agnosia that affects hearing, auditory sound agnosia, is broken into subdivisions based on level of processing impaired, and a semantic-associative form is investigated within the auditory agnosias.[2] Causes[edit]