BMJ
Professional/CME
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Targeting residual cardiovascular risk: raising high-density lipoprotein cholesterol levels -- Hausenloy and Yellon 94 (6): 706 -- Heart
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Management of dyslipidaemias -- Paramsothy and Knopp 92 (10): 1529 -- Heart
Peri-articular ‘hard tumours’ in soft tissues -- Varma et al. 75 (886): 492 -- Postgraduate Medical Journal
A 17-year-old man presented with painless, progressively increasing, non-simultaneous, peri-articular swellings evolving for the last 7 years. He had undergone repeated drainage of white chalky material and received adequate antitubercular therapy without any relief. He subsequently developed multiple non-healing sinuses. There was no history of colicky pain, graveluria, dyspnoea, excessive intake of antacids and vitamin D preparations, trauma or family history of similar disorder.
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Review: once daily LMWH is as effective as twice daily LMWH for initial treatment of venous thromboembolism -- 9 (2): 51 -- Evidence-Based Nursing
QUESTION: In patients with suspected venous thromboembolism (VTE) (ie, deep venous thrombosis [DVT] or pulmonary embolism [PE]), can a quantitative latex d -dimer assay rule out VTE? Design Blinded comparison of the results of the latex d -dimer test (MDA d -Dimer, Organon Teknika Corporation, Durham, NC, USA) with objective testing done at presentation and during follow up (3 mo). Setting 4 tertiary care university medical centres in Ontario, Canada. Patients
A negative quantitative latex d-dimer assay helped to rule out venous thromboembolism -- 6 (6): 188 -- Evidence-Based Medicine
Prophylaxis of venous thromboembolism in patients with lower limb plaster cast immobilisation -- Brown and Bleetman 24 (7): 495 -- Emergency Medicine Journal
The abstract/extract view of this article is free (link available in right hand box). Access to the full text of this article requires a subscription or payment. To view this item, select one of the options below:The renal tubule is responsible for the reabsorption of more than 99% of the water and sodium in the glomerular ultrafiltrate. Congenital or acquired tubular dysfunction can therefore readily cause profound electrolyte and volume disturbance. The tubule also has to regulate acid–base balance, mineral homoeostasis, and the excretion of organic anions and drugs.
Molecular developments in renal tubulopathies -- VAN'T HOFF 83 (3): 189 -- Archives of Disease in Childhood
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Peripheral blood culture contamination in adults and venepuncture technique: prospective cohort study -- Qamruddin et al. 61 (4): 509 -- Journal of Clinical Pathology
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Is it my calcium, Doctor? -- Stanley and Shaw 94 (6): 169 -- ADC - Education and Practice
Acute glomerulonephritis -- Vinen and Oliveira 79 (930): 206 -- Postgraduate Medical Journal
+ Author Affiliations Correspondence to: Professor David Oliveira, Department of Renal Medicine, St George’s Hospital Medical School, Cranmer Terrace, London SW17 0RE, UK; d.oliveira@sghms.ac.uk Received 17 May 2002 Accepted 5 November 2002 Abstract Glomerulonephritis is an important cause of renal failure thought to be caused by autoimmune damage to the kidney. While each type of glomerulonephritis begins with a unique initiating stimulus, subsequent common inflammatory and fibrotic events lead to a final pathway of progressive renal damage.