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Womb development: how much is influenced by genes vs. mother's lifestyle? Random Monoallelic Gene Expression Increases upon Embryonic Stem Cell Differentiation: Developmental Cell. To view the full text, please login as a subscribed user or purchase a subscription.

Random Monoallelic Gene Expression Increases upon Embryonic Stem Cell Differentiation: Developmental Cell

Click here to view the full text on ScienceDirect. Figure 1 Identification of Monoallelically Expressed Genes in ESCs and NPCs. 14-3-3 Proteins Regulate a Cell-Intrinsic Switch from Sonic Hedgehog-Mediated Commissural Axon Attraction to Repulsion after Midline Crossing. To view the full text, please login as a subscribed user or purchase a subscription.

14-3-3 Proteins Regulate a Cell-Intrinsic Switch from Sonic Hedgehog-Mediated Commissural Axon Attraction to Repulsion after Midline Crossing

Click here to view the full text on ScienceDirect. Figure 1 Smo Is Required for Proper Postcrossing Commissural Axon Guidance along the AP Axis. (101) 3-D imaging sheds light on Apert Syndrome... - Neuroscience Library/Bibliothèque des Neurosciences. Scientists Discover the Very First Hipster. You’ve seen the cartoon before: a fish hoisting itself up on land with its front fins, being greeted with some snarky sign like, “Evolve at your own risk,” or something similar.

Scientists Discover the Very First Hipster

This fish has become a meme, so much so that when the discovery of Tiktaalik roseae, a 375-million year old fossil that displays a mix of fish and tetrapod characters, was announced in 2006 by the lab I was working in at the University of Chicago, I thought I’d start a movement with the geekiest version of the Darwin fish ever: The world's geekiest version of the Darwin fish bumper sticker. Too bad it's WRONG! Well, wouldn’t ya know it, that pesky thing called progress has unseated the “front-wheel drive” version of life invading land and replaced it with something much more… well, hipster. More evidence is in, and Tiktaalik, it seems, was built like an old Volvo – with rear-wheel drive.

Human Development (ライディーン Rydeen- Yellow Magic Orchestra) Les cellules souches nerveuses - CellulesSouchesNeurales.pdf. Vidéo I : Migration neuronale. Architecture tissulaire de l'encéphale. Luminy: Biologie: Licence de Sciences de la Vie/Tronc Commun [S1-S3] - Sciences de la Vie/Biochimie [S4-S6] - Sciences de la Vie/Biologie Cellulaire [S4-S6] - Toddler brain scan gives language insight. 8 October 2013Last updated at 20:58 ET By Helen Briggs BBC News The left hand side of the brain has more myelin The brain has a critical window for language development between the ages of two and four, brain scans suggest.

Toddler brain scan gives language insight

Environmental influences have their biggest impact before the age of four, as the brain's wiring develops to process new words, say UK and US scientists. The research in The Journal of Neuroscience suggests disorders causing language delay should be tackled early. It also explains why young children are good at learning two languages. The scientists, based at King's College London, and Brown University, Rhode Island, studied 108 children with normal brain development between the ages of one and six.

Continue reading the main story. Toddler brain scan gives language insight. 2013/09/30 > BE Israël 92 > Une protéine pour polariser les neurones durant leur migration. L’épigénétique ou comment le cerveau change de la naissance à l’adolescence. (89) Lithium in the brain Experiments with... - Neuroscience Library/Bibliothèque des Neurosciences. Maternal and Offspring Pools of Osteocalcin Influence Brain Development and Functions. To view the full text, please login as a subscribed user or purchase a subscription.

Maternal and Offspring Pools of Osteocalcin Influence Brain Development and Functions

Click here to view the full text on ScienceDirect. Figure 1 Osteocalcin Affects the Biosynthesis of Neurotransmitters. (89) Bone Hormone Influences Brain Development and... - Neuroscience Library/Bibliothèque des Neurosciences. Researchers identify key brain systems affected by fragile X syndrome. Scott Hall Researchers at the Stanford University School of Medicine have identified several large-scale neural systems in the brain that appear to be impaired by fragile X syndrome, the most common form of inherited intellectual disability.

Researchers identify key brain systems affected by fragile X syndrome

The findings could help scientists devise treatments for the disorder, which is caused by a gene mutation on the X chromosome. People with fragile X syndrome display strikingly similar behavior to that of people with autism, including pronounced social awkwardness, repetitive actions, language impairment and a restricted range of interests. But while autism is diagnosed solely by assessing behavior, fragile X has a distinct physical cause that stems from blocked production of a particular protein needed for proper brain development. The syndrome typically becomes apparent in children by age 2 as they show delayed development of language abilities. The neural systems the researchers identified include the language, visuospatial and salience networks.

Faulty stem cell regulation may contribute to cognitive deficits associated with Down syndrome. La dyslexie pourrait se repérer dans les gènes. La dyslexie se caractérise par une difficulté de lecture.

La dyslexie pourrait se repérer dans les gènes

Environ 10 % des enfants seraient touchés. Pour être efficace, la prise en charge doit se faire le plus tôt possible. © murphyeppoon, Flickr, cc by nc sa 2.0. Le cerveau, un organe épigénomiquement à part. DÉVELOPPEMENT: Bébés singes et bébés humains, pas si différents ? Votre bébé ne fait pas ses nuits? C'est génétique! Experimental compound offers fragile X treatment - Highlight of the Month. <p>Please enable JavaScript in your browser.

Experimental compound offers fragile X treatment - Highlight of the Month

</p> Figure 1: Mice with fragile X syndrome (FXS) but expressing neither the PAK nor FMRP proteins display greatly improved symptoms. © iStockphoto/Thinkstock Fragile X syndrome (FXS) is the most common inherited form of autism and intellectual disability, characterized by hyperactivity, repetitive behaviors and seizures. Although there are a number of medications that can help individuals to cope with the behavioral and mental health issues associated with the syndrome, as yet there are no proven therapies that directly impact the root cause of the disease. The international research team, led by Susumu Tonegawa of the RIKEN–MIT Center for Neural Circuit Genetics (CNCG) at the Massachusetts Institute of Technology (MIT) in the United States, recently demonstrated the remarkable effectiveness of their promising compound—called FRAX486—in model FXS mice. A promising venture Reproduced from Ref. 1 © 2013 B. A look inside children's minds.

When young children gaze intently at something or furrow their brows in concentration, you know their minds are busily at work.

A look inside children's minds

But you're never entirely sure what they're thinking. Now you can get an inside look. Psychologists led by the University of Iowa for the first time have peered inside the brain with optical neuroimaging to quantify how much 3- and 4-year-old children are grasping when they survey what's around them and to learn what areas of the brain are in play. The study looks at "visual working memory," a core cognitive function in which we stitch together what we see at any given point in time to help focus attention.

Identification Of Important Genomic Variations Will Likely Impact Down Syndrome. De la peau aux neurones sans passer par les cellules souches. Par Agnès Roux, Futura-Sciences Une équipe américaine a mis au point une nouvelle technologie permettant la transformation directe de cellules de la peau en neurones . Cette découverte prometteuse est un pas de plus vers le traitement des maladies neurologiques telles qu’ Alzheimer ou Parkinson. Dans le domaine de la recherche sur les cellules souches , les progrès avancent à grands pas. Ces cellules particulières sont capables de se multiplier indéfiniment et de se transformer en cellules spécialisées, comme des neurones ou des globules rouges . Elles peuvent ainsi être utilisées en thérapie cellulaire pour remplacer les tissus endommagés et pour soigner les maladies dégénératives comme Alzheimer .

Cependant, l’utilisation de cellules souches , qui proviennent majoritairement d’ embryons , pose de nombreux problèmes éthiques et politiques. Un gène expliquerait pourquoi les animaux sont sortis des eaux › Génétique. Le Mesosaurus, un reptile marin vivant il y a 300 millions d'années environ, soit peu après la sortie des eaux des premiers vertébrés marins, était muni de membres qui lui permettaient de se déplacer difficilement sur la terre ferme.

Un gène expliquerait pourquoi les animaux sont sortis des eaux › Génétique

Crédits : Kevmin Pourquoi les vertébrés sont-ils sortis de l'eau il y a 350 millions d'années ? Selon des chercheurs espagnols, cet évènement aurait été causé par la surexpression soudaine d'un gène, appelé Hox13d.