Group 1. Group 2. Group 3. Group 4. Yeast is a Cause of Cancer and Turmeric Can Kill Both, Research Confirms. 5th February 2016 By Sayer Ji Contributing Writer for Wake Up World A new study validates a controversial cancer theory, namely, that yeast in our body can contribute to not just feeding but actually causing cancer.
Can the ancient healing spice turmeric come to the rescue? A recent study published in Critical Reviews in Microbiology lends support to the concept that opportunistic Candida albicans (yeast) infection may not just be a consequence of cancer, but is an actively contributing cause as well. Titled, “Candida albicans and cancer: Can this yeast induce cancer development or progression? “There is currently increasing concern about the relation between microbial infections and cancer.
The four distinct ways by which Candida albicans may contribute to cancer are explained in more detail below: Production of carcinogenic byproducts: First, Candida Albicans produces nitrosamines, which are carcinogens that activate specific proto-oncogenes that could trigger cancerous lesions. Curcumin suppresses proliferation and induces... [Carcinogenesis. 2011.
Two curcumin analogs squash the growth of multiple myeloma cells… On November 26 I received a Google Alert about an “Anticancer Research” November 2011 abstract discussing not just one but TWO curcumin analogs, GO-YO30 and GO-YO78, which have shown much stronger growth-suppressive activity against myeloma cells than plain old curcumin: They also very strongly suppressed many bothersome (in cancer) pathways, including our old nemesis, NF-kappaB, which is very much involved in the well-being of myeloma cells…again, much more effectively than regular curcumin…up to 15 times more!
I tried to get my hands on the full study, but without any luck. And that is mainly why I didn’t finish my post last week, leaving it in draft form. Oh okay, it’s time now to fess up: I totally FORGOT about it. Until yesterday, which is when several attentive blog readers (thanks!!!) Sent me messages about these two analogs. Yes, of course, these tests were carried out on CELLS, not on humans. Redox modulation and human bile duct cancer inhibition by curcumin. A phase I study investigating the safety and pharmacokinetics of highly bioavailable curcumin in cancer patients.
Abstract: Curcumin Inhibits Cholangiocarcinoma Cell Growth by Modulation of the Cellular Redox Status (3rd Asian Pacific Regional ISSX Meeting) Effect of Curcumin on Opisthorchiasis and Cholangiocarcinoma in Animal Models. Margaret’s curcumin PROTOCOL. May 17 2007 post.
Here it is, finally. I have been procrastinating about posting the protocol on my blog, even though I have written it out in countless private e-mails, mainly because I felt I should add a disclaimer, and, to be honest, I didn’t want to do that. The only reason I decided I needed to put a disclaimer here is because I have seen other alternative cancer treatment blogs do it. So, first, let’s get the disclaimer out of the way: my blog presents information based on my own experience and research.
I do NOT intend to tell others what to do, or to become a substitute for anybody’s healthcare provider. Phew. Copub - Abstracts with co-occurrences of curcumin and cholangiocarcinoma. Anticancer activities against cholangiocarcinoma, toxicity and pharmacological activities of Thai medicinal plants in animal models. Background Chemotherapy of cholangiocarcinoma (CCA), a devastating cancer with increasing worldwide incidence and mortality rates, is largely ineffective.
The discovery and development of effective chemotherapeutics is urgently needed. Methods/Design The study aimed at evaluating anticancer activities, toxicity, and pharmacological activities of the curcumin compound (CUR), the crude ethanolic extracts of rhizomes of Zingiber officinale Roscoe (Ginger: ZO) and Atractylodes lancea thung. DC (Khod-Kha-Mao: AL), fruits of Piper chaba Hunt. Results Promising anticancer activity against CCA in nude mouse xenograft model was shown for the ethanolic extract of AL at all oral dose levels (1000, 3000, and 5000 mg/kg body weight) as well as the extracts of ZO, PPF, and CUR compound at the highest dose level (5000, 4000, and 5000 mg/kg body weight, respectively).
Conclusion. Posts about Cholangiocarcinoma and Curcumin. Last modified: April 24, 2013 By using or accessing (The Website) you hereby affirm you understand and agree to the following: This agreement is between you and Treato Ltd.
Sensitivity of Cholangiocarcinoma Cells to Chemotherapeutic Agents and Curcumin. Biliary excretion of curcumin is mediated by... [Biol Pharm Bull. 2012. Curcumin decreases cholangiocarcinogenesis in hamsters by suppressing inflammation-mediated molecular events related to multistep carcinogenesis - Prakobwong - 2010 - International Journal of Cancer. Abstract Cholangiocarcinoma (CCA) is a highly metastatic tumor linked to liver fluke infection and consumption of nitrosamine-contaminated foods and is a major health problem especially in South-Eastern Asia.
In search for a suitable chemopreventive agents, we investigated the effect of curcumin, a traditional anti-inflammatory agent derived from turmeric (Curcuma longa), on CCA development in an animal model by infection with the liver fluke Opisthorchis viverrini and administration of N-nitrosodimethylamine and fed with curcumin-supplemented diet. Audio - A Simple Spice that may Battle Cancer - Johns Hopkins Medicine Media Player. Curcumin suppresses proliferation and induces apoptosis in human biliary cancer cells through modulation of multiple cell signaling pathways. Abstract: Curcumin Inhibits Cholangiocarcinoma Cell Growth by Modulation of the Cellular Redox Status (3rd Asian Pacific Regional ISSX Meeting) Curcumin suppresses proliferation and induces... [Carcinogenesis. 2011.